Acute glomerulonephritis induced hypertension is more common clinical hypertension secondary one. Its characteristics is that children with mild and high blood pressure are more rare, or transient, and mainly to systolic blood pressure, while the incidence of hypertension in adult patients was 70% to 80%, systolic and diastolic blood pressure are often increased. The degree of blood pressure, mostly mild to moderate blood pressure increased significantly only in the elderly and terminally ill. From the time point of view, the emergence of high blood pressure more in edema, proteinuria appeared at the same time, only a small number of patients before other symptoms of high blood pressure. Varying the duration of hypertension, edema and generally parallel the changes in urine, the vast majority showed a slow decline, a few sharp rise in blood pressure may even lead to hypertensive encephalopathy and heart failure . Pediatric patients, as well as to the performance of hypertensive encephalopathy as the first who should be in the diagnosis of attention.
Acute glomerulonephritis induced hypertension causes renal disease is currently thought to cause sodium and water excretion disorders, resulting in volume status and renal disease may result in increased secretion of the kidney boost substances. The diagnosis is generally based on the following points:
(1) children or young patients, more common to 5 to 20 years old.
(2) prior to the onset more than a history of streptococcal infection and a history of viral infection and often accompanied by fever, edema , hematuria , severe heart failure or hypertensive encephalopathy can occur.
(3) urinalysis proteinuria, red blood cells and casts.
(4) hypertension occurred in oliguria or swelling of the swelling subsided, the blood pressure is lower.
(5) systolic and diastolic blood pressure were increased, systolic blood pressure more in the 17.3 ~ 22.7kPa (130 ~ 170mmHg) between.
(6) have high blood pressure caused by dizziness , headache , tinnitus , nausea and other symptoms.
